BRCA2 promotes DNA‐RNA hybrid resolution by DDX5 helicase at DNA
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Descrição
Role of the DEAD-box RNA helicase DDX5 (p68) in cancer DNA repair, immune suppression, cancer metabolic control, virus infection promotion, and human microbiome (microbiota) negative influence
Sources, resolution and physiological relevance of R-loops and RNA–DNA hybrids
RAD51-mediated R-loop formation acts to repair transcription-associated DNA breaks driving antigenic variation in Trypanosoma brucei
Frontiers Making Connections: Integrative Signaling Mechanisms Coordinate DNA Break Repair in Chromatin
DDX5 plays a central role in resolving DNA replication /
Role of the DEAD-box RNA helicase DDX5 (p68) in cancer DNA repair, immune suppression, cancer metabolic control, virus infection promotion, and human microbiome (microbiota) negative influence
PDF] DNA-RNA hybrids at DSBs interfere with repair by homologous recombination
BRCA2 promotes DNA‐RNA hybrid resolution by DDX5 helicase at DNA breaks to facilitate their repair‡
BRCA2 promotes DNA‐RNA hybrid resolution by DDX5 helicase at DNA breaks to facilitate their repair‡
RNA:DNA hybrids from Okazaki fragments contribute to establish the Ku-mediated barrier to replication-fork degradation - ScienceDirect
Hypoxia-mediated regulation of DDX5 through decreased chromatin accessibility and post-translational targeting restricts R-loop accumulation
Looping out of control: R-loops in transcription-replication conflict
Cancers, Free Full-Text
BRCA2 promotes DNA‐RNA hybrid resolution by DDX5 helicase at DNA breaks to facilitate their repair‡
ZFP281-BRCA2 prevents R-loop accumulation during DNA replication
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