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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis

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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Protective effect of dexmedetomidine on vascular permeability of
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PDF] Sepsis and endothelial permeability.
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Neutrophil, neutrophil extracellular traps and endothelial cell
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate induces vascular permeability via disruption of VE
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PDHA1 hyperacetylation-mediated lactate overproduction promotes
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
IJMS, Free Full-Text
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Endothelial barrier dysfunction in systemic inflammation is
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Microorganisms, Free Full-Text
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Vascular Endothelial-Cadherin Stabilizes at Cell–Cell Junctions by
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate promotes endothelial-to-mesenchymal transition via Snail1
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Endothelial barrier dysfunction in systemic inflammation is
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Endothelial damage in septic shock patients as evidenced by
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Protein Interactions at Endothelial Junctions and Signaling
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