Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming
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Changing POU dimerization preferences converts Oct6 into a pluripotency inducer
In silico identification of natural product inhibitors against Octamer-binding transcription factor 4 (Oct4) to impede the mechanism of glioma stem cells
Regulation of Oct4 in stem cells and neural crest cells - Patel - 2022 - Birth Defects Research - Wiley Online Library
Evolutionary origin of vertebrate OCT4/POU5 functions in supporting pluripotency
A unique Oct4 interface is crucial for reprogramming to pluripotency
A unique Oct4 interface is crucial for reprogramming to pluripotency
Reprogramming enriches for somatic cell clones with small-scale mutations in cancer-associated genes - ScienceDirect
Cancers, Free Full-Text
Changing POU dimerization preferences converts Oct6 into a pluripotency inducer
Molecular Reproduction & Development, Reproductive Biology Journal
Evolutionary Origin of Vertebrate OCT4/POU5 Functions in Supporting Pluripotency
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Changing POU dimerization preferences converts Oct6 into a pluripotency inducer. - Abstract - Europe PMC
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